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Table 4 Comparison between improved and deceased groups regarding categorical variables

From: Utility of prognostic scores in predicting short-term mortality in patients with acute-on-chronic liver failure

 

Improved (n=64)

Deceased (n=185)

P

Sex

Males

47 (73)

130 (70.3)

0.678

Females

17 (27)

55 (29.7)

Precipitating factor of ACLF

Sepsis

16 (25)

108 (58.4)

<0.0001

Upper GI bleeding

7 (10.9)

18 (9.7)

De novo AIH

1 (1.6)

0 (0)

AIH flare

6 (9.4)

2 (1.1)

DILI

7 (10.9)

2 (1.1)

HBV

1 (1.6)

2 (1.1)

HCV

0 (0)

1 (0.5)

HDV

1 (1.6)

1 (0.5)

HEV

0 (0)

2 (1.1)

Wilson’s disease

1 (1.6)

0 (0)

Unknown

24 (37.5)

47 (25.4)

Etiology of pre-existing CLD

HCV

30 (46.8)

113 (61.1)

0.055

AIH

6 (9.4)

2 (1.1)

Budd-Chiari syndrome

0 (0)

2 (1.1)

HBV

5 (7.8)

8 (4.3)

HBV-HCV co-infection

0 (0)

1 (0.5)

Unknown

23 (35.9)

59 (31.9)

Ascites

No

37 (57.8)

38 (20.5)

<0.0001

Mild to moderate

24 (37.5)

131 (70.8)

Marked

3 (4.7)

16 (8.6)

Hepatic encephalopathy

No

56 (87.5)

26 (14.1)

<0.0001

Grades I–II

7 (10.9)

116 (62.7)

Grades III–IV

1 (1.6)

43 (23.2)

Initial admission

Ward

50 (78.1)

8 (4.3)

<0.0001

ICU

14 (21.9)

177 (95.7)

Child-Pugh class

B

16 (23.8)

3 (1.6)

<0.0001

C

48 (76.2)

182 (98.4)

ACLF grade

1

51 (79.7)

39 (21.1)

<0.0001

2

13 (20.3)

95 (51.4)

3

0 (0)

51 (27.6)

  1. ACLF Acute-on-chronic liver failure, AIH Autoimmune hepatitis, DILI Drug-induced liver injury, ICU Intensive care unit, GI Gastrointestinal, HBV Hepatitis B virus, HCV Hepatitis C virus, HDV Hepatitis D virus, HEV Hepatitis E virus